Team:Paris/Modeling/f2

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Revision as of 15:55, 1 August 2008

No particular strain.

Exactly as for the binding of IPTG on LacR, leading to a lowered inhibition of p-lac, aTc binds to TetR. So, after having determinated the Hill function [expr.p-tet] = ƒ2(TetR) = ƒ2aux(IPTG), we can study [expr.Ptet] = ƒ2ter([TetR],[aTc]), and determine by calcul [TetR_active] = ƒ2bis(aTc) = f2^-1([expr.Ptet]), to endly give an analytical expression of ƒ2ter = ƒ2οƒ2bis

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	[[Image:f2.png]]		[[Image:f2.png]]

-	Exactly as for the binding of IPTG on LacR, leading to a lowered inhibition of p-lac, aTc binds to TetR. So, after having determinated the Hill function [expr.p-tet] = 2(TetR) = 2aux(IPTG), we can study [expr.''Ptet''] = 2ter([TetR],[aTc]), and determine by calcul [TetR<sub>active<sub>] = 2bis(aTc) = f2<sup>-1</sup>([expr.''Ptet'']), to endly give an analytical expression of 2ter = 2&#~~186~~;2bis	+	Exactly as for the binding of IPTG on LacR, leading to a lowered inhibition of p-lac, aTc binds to TetR. So, after having determinated the Hill function [expr.p-tet] = 2(TetR) = 2aux(IPTG), we can study [expr.''Ptet''] = 2ter([TetR],[aTc]), and determine by calcul [TetR<sub>active</sub>] = 2bis(aTc) = f2<sup>-1</sup>([expr.''Ptet'']), to endly give an analytical expression of 2ter = 2ο2bis